A novel mechanism for imatinib mesylate-induced cell death of BCR-ABL-positive human leukemic cells: caspase-independent, necrosis-like programmed cell death mediated by serine protease activity.

Masayuki Okada; Souichi Adachi; Tsuyoshi Imai; Ken-ichiro Watanabe; Shin-ya Toyokuni; Masaki Ueno; Antonis S Zervos; Guido Kroemer; Tatsutoshi Nakahata

doi:10.1182/blood-2003-05-1605

A novel mechanism for imatinib mesylate-induced cell death of BCR-ABL-positive human leukemic cells: caspase-independent, necrosis-like programmed cell death mediated by serine protease activity.

Identifier:nobleid.org/w1/20260515/1AF97281

Type:Journal Article

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A novel mechanism for imatinib mesylate-induced cell death of BCR-ABL-positive human leukemic cells: caspase-independent, necrosis-like programmed cell death mediated by serine protease activity.

Identifier:nobleid.org/w1/20260515/1AF97281

Type:Journal Article

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