AML1/RUNX1 mutations are infrequent, but related to AML-M0, acquired trisomy 21, and leukemic transformation in pediatric hematologic malignancies.

Takeshi Taketani; Tomohiko Taki; Junko Takita; Masahiro Tsuchida; Ryoji Hanada; Teruaki Hongo; Takashi Kaneko; Atsushi Manabe; Kohmei Ida; Yasuhide Hayashi

doi:10.1002/gcc.10246

AML1/RUNX1 mutations are infrequent, but related to AML-M0, acquired trisomy 21, and leukemic transformation in pediatric hematologic malignancies.

Identifier:nobleid.org/w1/20260515/EAADE358

Type:Journal Article

0 views

Support unavailableClaim Your Authorship

View Original Paper Source

Embeddable Badge

[![NobleID](https://www.nobleid.org/api/badge/EAADE358.svg?ark=w1%2F20260515%2FEAADE358)](https://nobleid.org/work/w1/20260515/EAADE358)

ARK Inflections

Metadata Policy

Metadata Formats

Bibliometric Analysis

Impact metrics, research fronts, co-authorship networks →

Authors & Claims

Paper Authors

Claim authorship of this work

AML1/RUNX1 mutations are infrequent, but related to AML-M0, acquired trisomy 21, and leukemic transformation in pediatric hematologic malignancies.

Identifier:nobleid.org/w1/20260515/EAADE358

Type:Journal Article

0 views

Support unavailableClaim Your Authorship

View Original Paper Source

Embeddable Badge

[![NobleID](https://www.nobleid.org/api/badge/EAADE358.svg?ark=w1%2F20260515%2FEAADE358)](https://nobleid.org/work/w1/20260515/EAADE358)

ARK Inflections

Metadata Policy

Metadata Formats

Bibliometric Analysis

Impact metrics, research fronts, co-authorship networks →

Authors & Claims

Paper Authors

Claim authorship of this work