Inhibition of ataxia telangiectasia mutated (ATM) prevents the prolonged increase in phosphorylation of Akt substrate of 160 kDa (AS160) subsequent to activation of the AMP‐activated protein kinase (AMPK)

Lewis, LaQuinta R.; Davis, Jennifer; Akinola, Ayotola; Fisher, Jonathan S.

doi:10.1096/fasebj.23.1_supplement.782.2

Inhibition of ataxia telangiectasia mutated (ATM) prevents the prolonged increase in phosphorylation of Akt substrate of 160 kDa (AS160) subsequent to activation of the AMP‐activated protein kinase (AMPK)

LaQuinta R. Lewis, Jennifer Davis, Ayotola Akinola, Jonathan S. Fisher

LaQuinta R. Lewis

NobleIDNI4P44W36R67S55

Type:Journal Article

Claim Your Authorship

Sign in to see the full record

Creating an account is free.

Create an account

For data access, contact info@nobleid.org.

Inhibition of ataxia telangiectasia mutated (ATM) prevents the prolonged increase in phosphorylation of Akt substrate of 160 kDa (AS160) subsequent to activation of the AMP‐activated protein kinase (AMPK) | NobleID